H2forLife: Supporting lower oxidative stress levels during viral infections

Virus-induced actions are associated with oxidative stress and cellular damage. (1) Viruses can effect the cell pro-antioxidant balance by increasing cellular pro-oxidants free radicals and inhibiting the synthesis of antioxidant enzymes. Viral infections have shown that they can trigger the immune system to create cascading release of pro-inflammatory cytokines ("cytokine storm") which can lead to organ failure. (2)

In addition, oxidative stress is associated with Adult Respiratory Distress Syndrome (ARDS). ARDS is a problem in intensive care units (ICU's) because of it's frequency and low survival rate of less that 50%. The problem is compounded during this epidemic as it necessitates high concentrations of O2 delivered by mechanical ventilators in an ICU. (3)

H2forLife significantly reduces oxidative stress, as measured and verified by Oxicheck (an oxidative stress measurement device), in H2forLife (H2FL) customers from 2017-2019. What is significant for viral infections is the speed. Infections like influenza, COVID-19 and malaria trigger massive body aches and high levels of oxidative stress (OS). Measured OS tests in 2017-2018 of many H2FL customers before and after drinking H2forLife has shown that it acts quickly to reduce oxidative stress as much as 50% in 30 minutes.

Infected patients drinking H2forLife are reporting pain reduction levels that last up to 4-5 hours signalling a lowering of oxidative stress. Can these lower levels also prevent hospitilizations? That we will need to answer in the coming days with more data from H2FL customers. We are asking all customers who have tested positive to let us know. We will send out a medical survey to allow us to report to public health agencies and research groups.


(1) https://www.researchgate.net/profile/Kathleen_Schwarz/publication/222319272_Oxidative_stress_during_viral_infection_A_review/links/5ab970e6aca2722b97d121a9/Oxidative-stress-during-viral-infection-A-review.pdf

(2) https://link.springer.com/content/pdf/10.1186/s40779-020-00240-0.pdf)

(3) https://link.springer.com/chapter/10.1007%2F978-1-4684-5730-8_69

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